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Moreover, cyclophosphamide (CTX) induces increased apoptosis and reduced cellular proliferation of GCs, and metformin can exert anti-apoptotic and cell proliferative effects against chemotoxicity via the AMPK-dependent p53 signaling pathway (Huang et al., 2021)

However, the relationships between levels of TSH, free T4 (FT4) and free T3 (FT3) are not the same in LT4-treated patients as in healthy, euthyroid individuals, and the distribution, metabolism and excretion of exogenous LT4 differ from those of endogenous T4 [23]

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